What part of the brain is responsible for antisocial personality disorder?

What part of the brain is responsible for antisocial personality disorder?

Key regions commonly found to be impaired in antisocial populations include the prefrontal cortex (particularly orbitofrontal and dorsolateral prefrontal cortex), superior temporal gyrus, amygdala-hippocampal complex, and anterior cingulate cortex.

What therapy is best for ASPD?

Psychotherapy. Psychotherapy, also called talk therapy, is sometimes used to treat antisocial personality disorder. Therapy may include, for example, anger and violence management, treatment for alcohol or substance misuse, and treatment for other mental health conditions.

Does CBT work for ASPD?

For people with ASPD, CBT may help them think about how their behavior affects others and gets them into trouble. CBT is offered as individual therapy, group therapy, and/or family therapy.

What happens to the brain in ASPD?

Emerging neuroimaging research suggests that ASPD is linked with abnormal brain anatomy. Raine et al. found that the prefrontal gray matter volume in ASPD was reduced by about 11%, in comparison to that of the control group3.

How does antisocial disorder affect the brain?

MRI scans on the brains of 672 people with lifelong antisocial behavior showed a particular thinning of cortex and reduced cortical surface area in parts of the brain responsible for thinking, motivation and controlling emotions, according to research just published in The Lancet Psychiatry.

Can ASPD be cured?

There is no cure for antisocial personality disorder. People generally manage the condition throughout their lives. But medication and therapy can help you cope with certain aspects of the disorder. The right treatment may help you adjust your behavior and reduce harm to those around you.

What part of the brain is affected by personality disorders?

the amygdala – which plays an important role in regulating emotions, especially the more “negative” emotions, such as fear, aggression and anxiety.